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hep3b cells  (CLS Cell Lines Service GmbH)


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    Structured Review

    CLS Cell Lines Service GmbH hep3b cells
    Hep3b Cells, supplied by CLS Cell Lines Service GmbH, used in various techniques. Bioz Stars score: 90/100, based on 8 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/hep3b+cells/HEP3B+Cells/custom%40305141%4010%2E64898%2F2026%2E05%2E15%2E725527
    Average 90 stars, based on 8 article reviews
    hep3b cells - by Bioz Stars, 2026-10
    90/100 stars

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    Expressing:

    Article Title: Pharmacological inhibition of deubiquitinase UCH-L1 by LDN57444 sensitises hepatocellular carcinoma to sorafenib by reverting drug-induced adaptive responses
    Article Snippet: PrePrint: to quantify UCH-L1 expression reported as log2 fold changes with Benjamini–Hochberg adjusted p-values to control the FDR. The human liver cancer cell line Hep 3B2.1-7 (ATCC HB-8064) was purchased from Cytion (Eppelheim, Germany). SNU-423 (CRL-2238) liver cancer cells were obtained from ATCC. Hep3B cells were cultured in complete DMEM, i.e. Dulbecco’s modified Eagle’s medium (DMEM) supplemented with

    Modification:

    Article Title: Pharmacological inhibition of deubiquitinase UCH-L1 by LDN57444 sensitises hepatocellular carcinoma to sorafenib by reverting drug-induced adaptive responses
    Article Snippet: PrePrint: to quantify UCH-L1 expression reported as log2 fold changes with Benjamini–Hochberg adjusted p-values to control the FDR. The human liver cancer cell line Hep 3B2.1-7 (ATCC HB-8064) was purchased from Cytion (Eppelheim, Germany). SNU-423 (CRL-2238) liver cancer cells were obtained from ATCC. Hep3B cells were cultured in complete DMEM, i.e. Dulbecco’s modified Eagle’s medium (DMEM) supplemented with

    Cell Culture:

    Article Title: Pharmacological inhibition of deubiquitinase UCH-L1 by LDN57444 sensitises hepatocellular carcinoma to sorafenib by reverting drug-induced adaptive responses
    Article Snippet: PrePrint: to quantify UCH-L1 expression reported as log2 fold changes with Benjamini–Hochberg adjusted p-values to control the FDR. The human liver cancer cell line Hep 3B2.1-7 (ATCC HB-8064) was purchased from Cytion (Eppelheim, Germany). SNU-423 (CRL-2238) liver cancer cells were obtained from ATCC. Hep3B cells were cultured in complete DMEM, i.e. Dulbecco’s modified Eagle’s medium (DMEM) supplemented with

    Control:

    Article Title: Pharmacological inhibition of deubiquitinase UCH-L1 by LDN57444 sensitises hepatocellular carcinoma to sorafenib by reverting drug-induced adaptive responses
    Article Snippet: PrePrint: to quantify UCH-L1 expression reported as log2 fold changes with Benjamini–Hochberg adjusted p-values to control the FDR. The human liver cancer cell line Hep 3B2.1-7 (ATCC HB-8064) was purchased from Cytion (Eppelheim, Germany). SNU-423 (CRL-2238) liver cancer cells were obtained from ATCC. Hep3B cells were cultured in complete DMEM, i.e. Dulbecco’s modified Eagle’s medium (DMEM) supplemented with

    Staining:

    Article Title: Pharmacological inhibition of deubiquitinase UCH-L1 by LDN57444 sensitises hepatocellular carcinoma to sorafenib by reverting drug-induced adaptive responses
    Article Snippet: PrePrint: to quantify UCH-L1 expression reported as log2 fold changes with Benjamini–Hochberg adjusted p-values to control the FDR. The human liver cancer cell line Hep 3B2.1-7 (ATCC HB-8064) was purchased from Cytion (Eppelheim, Germany). SNU-423 (CRL-2238) liver cancer cells were obtained from ATCC. Hep3B cells were cultured in complete DMEM, i.e. Dulbecco’s modified Eagle’s medium (DMEM) supplemented with

    High Content Screening:

    Article Title: Pharmacological inhibition of deubiquitinase UCH-L1 by LDN57444 sensitises hepatocellular carcinoma to sorafenib by reverting drug-induced adaptive responses
    Article Snippet: PrePrint: to quantify UCH-L1 expression reported as log2 fold changes with Benjamini–Hochberg adjusted p-values to control the FDR. The human liver cancer cell line Hep 3B2.1-7 (ATCC HB-8064) was purchased from Cytion (Eppelheim, Germany). SNU-423 (CRL-2238) liver cancer cells were obtained from ATCC. Hep3B cells were cultured in complete DMEM, i.e. Dulbecco’s modified Eagle’s medium (DMEM) supplemented with



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    SLC41A3 promotes HCC progression. (A) Western blot analysis confirming the knockdown efficiency of SLC41A3 in <t>Hep3B</t> and HuH7 cells transfected with shRNA targeting SLC41A3. (B) CCK-8 assay evaluating cell proliferation at specified time points after SLC41A3 silencing in HCC cells. (C) Colony formation ability of Hep3B and HuH7 cells was inhibited after SLC41A3 knockdown. (D) Transwell migration and invasion assays indicated impaired migration and invasion capabilities after SLC41A3 downregulation in both cell lines. (E) Wound healing assay showed reduced cell motility after SLC41A3 knockdown. Scale bar, 100 μm. Data are presented as the mean ± SD of three independent experiments. *** P < 0.001.
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    ATCC human hepatoma cell line hep3b
    SLC41A3 promotes HCC progression. (A) Western blot analysis confirming the knockdown efficiency of SLC41A3 in <t>Hep3B</t> and HuH7 cells transfected with shRNA targeting SLC41A3. (B) CCK-8 assay evaluating cell proliferation at specified time points after SLC41A3 silencing in HCC cells. (C) Colony formation ability of Hep3B and HuH7 cells was inhibited after SLC41A3 knockdown. (D) Transwell migration and invasion assays indicated impaired migration and invasion capabilities after SLC41A3 downregulation in both cell lines. (E) Wound healing assay showed reduced cell motility after SLC41A3 knockdown. Scale bar, 100 μm. Data are presented as the mean ± SD of three independent experiments. *** P < 0.001.
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    ATCC cell lines hep3b atcc hb 8064 hepg2 atcc hb 8065 oligonucleotides primers
    SLC41A3 promotes HCC progression. (A) Western blot analysis confirming the knockdown efficiency of SLC41A3 in <t>Hep3B</t> and HuH7 cells transfected with shRNA targeting SLC41A3. (B) CCK-8 assay evaluating cell proliferation at specified time points after SLC41A3 silencing in HCC cells. (C) Colony formation ability of Hep3B and HuH7 cells was inhibited after SLC41A3 knockdown. (D) Transwell migration and invasion assays indicated impaired migration and invasion capabilities after SLC41A3 downregulation in both cell lines. (E) Wound healing assay showed reduced cell motility after SLC41A3 knockdown. Scale bar, 100 μm. Data are presented as the mean ± SD of three independent experiments. *** P < 0.001.
    Cell Lines Hep3b Atcc Hb 8064 Hepg2 Atcc Hb 8065 Oligonucleotides Primers, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    ATCC cell culture hep3b cells
    SLC41A3 promotes HCC progression. (A) Western blot analysis confirming the knockdown efficiency of SLC41A3 in <t>Hep3B</t> and HuH7 cells transfected with shRNA targeting SLC41A3. (B) CCK-8 assay evaluating cell proliferation at specified time points after SLC41A3 silencing in HCC cells. (C) Colony formation ability of Hep3B and HuH7 cells was inhibited after SLC41A3 knockdown. (D) Transwell migration and invasion assays indicated impaired migration and invasion capabilities after SLC41A3 downregulation in both cell lines. (E) Wound healing assay showed reduced cell motility after SLC41A3 knockdown. Scale bar, 100 μm. Data are presented as the mean ± SD of three independent experiments. *** P < 0.001.
    Cell Culture Hep3b Cells, supplied by ATCC, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/hep3b+cells/Hep+3B2%2E1-7/pm41974280-54-0-4
    Average 96 stars, based on 1 article reviews
    cell culture hep3b cells - by Bioz Stars, 2026-10
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    Image Search Results


    SLC41A3 promotes HCC progression. (A) Western blot analysis confirming the knockdown efficiency of SLC41A3 in Hep3B and HuH7 cells transfected with shRNA targeting SLC41A3. (B) CCK-8 assay evaluating cell proliferation at specified time points after SLC41A3 silencing in HCC cells. (C) Colony formation ability of Hep3B and HuH7 cells was inhibited after SLC41A3 knockdown. (D) Transwell migration and invasion assays indicated impaired migration and invasion capabilities after SLC41A3 downregulation in both cell lines. (E) Wound healing assay showed reduced cell motility after SLC41A3 knockdown. Scale bar, 100 μm. Data are presented as the mean ± SD of three independent experiments. *** P < 0.001.

    Journal: Frontiers in Immunology

    Article Title: Comprehensive characterization of SLC41A3 identifies it as an immune-related prognostic biomarker and therapeutic target in hepatocellular carcinoma

    doi: 10.3389/fimmu.2026.1861310

    Figure Lengend Snippet: SLC41A3 promotes HCC progression. (A) Western blot analysis confirming the knockdown efficiency of SLC41A3 in Hep3B and HuH7 cells transfected with shRNA targeting SLC41A3. (B) CCK-8 assay evaluating cell proliferation at specified time points after SLC41A3 silencing in HCC cells. (C) Colony formation ability of Hep3B and HuH7 cells was inhibited after SLC41A3 knockdown. (D) Transwell migration and invasion assays indicated impaired migration and invasion capabilities after SLC41A3 downregulation in both cell lines. (E) Wound healing assay showed reduced cell motility after SLC41A3 knockdown. Scale bar, 100 μm. Data are presented as the mean ± SD of three independent experiments. *** P < 0.001.

    Article Snippet: The human HCC cell lines Hep3B and Huh7 were purchased from Procell Biotechnology.

    Techniques: Western Blot, Knockdown, Transfection, shRNA, CCK-8 Assay, Migration, Wound Healing Assay